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Ca(2+)-dependent nitric oxide release in the injured endothelium of excised rat aorta: a promising mechanism applying in vascular prosthetic devices in aging patients

BACKGROUND: Nitric oxide is key to endothelial regeneration, but it is still unknown whether endothelial cell (EC) loss results in an increase in NO levels at the wound edge. We have already shown that endothelial damage induces a long-lasting Ca(2+ )entry into surviving cells though connexin hemich...

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Detaylı Bibliyografya
Asıl Yazarlar: Berra-Romani, Roberto, Avelino-Cruz, José Everardo, Raqeeb, Abdul, Della Corte, Alessandro, Cinelli, Mariapia, Montagnani, Stefania, Guerra, Germano, Moccia, Francesco, Tanzi, Franco
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: BioMed Central 2013
Konular:
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC3851245/
https://ncbi.nlm.nih.gov/pubmed/24266895
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1471-2482-13-S2-S40
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