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Selenoprotein P inhibits radiation-induced late reactive oxygen species accumulation and normal cell injury
PURPOSE: Radiation is a common mode of cancer therapy whose outcome is often limited due to normal tissue toxicity. We have shown previously that radiation-induced late reactive oxygen species (ROS) accumulation precedes cell death, suggesting that metabolic oxidative stress could regulate cellular...
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| 主要な著者: | , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2013
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3787541/ https://ncbi.nlm.nih.gov/pubmed/24074935 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.ijrobp.2013.06.2063 |
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