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Correction of metabolic abnormalities in a rodent model of obesity, metabolic syndrome, and type 2 diabetes mellitus by inhibitors of hepatic protein kinase C-ι

Excessive activity of hepatic atypical protein kinase (aPKC) is proposed to play a critical role in mediating lipid and carbohydrate abnormalities in obesity, the metabolic syndrome, and type 2 diabetes mellitus. In previous studies of rodent models of obesity and type 2 diabetes mellitus, adenovira...

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Bibliografski detalji
Glavni autori: Sajan, Mini P., Nimal, Sonali, Mastorides, Stephen, Acevedo-Duncan, Mildred, Kahn, C. Ronald, Fields, Alan P., Braun, Ursula, Leitges, Michael, Farese, Robert V.
Format: Artigo
Jezik:Inglês
Izdano: 2012
Teme:
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC3786325/
https://ncbi.nlm.nih.gov/pubmed/22225955
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.metabol.2011.12.008
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