Disruption of TAB1/p38α Interaction Using a Cell-permeable Peptide Limits Myocardial Ischemia/Reperfusion Injury

Targeting the adaptor protein (transforming growth factor-β (TGF-β)-activated protein kinase 1 (TAK1)-binding protein 1) (TAB1)-mediated non-canonical activation of p38α to limit ischemia/reperfusion (I/R) injury after an acute myocardial infarction seems to be attractive since TAB1/p38α interaction...

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Detalhes bibliográficos
Main Authors: Wang, Qingyang, Feng, Jiannan, Wang, Jing, Zhang, Xueying, Zhang, Dalin, Zhu, Ting, Wang, Wendie, Wang, Xiaoqian, Jin, Jianfeng, Cao, Junxia, Li, Xinying, Peng, Hui, Li, Yan, Shen, Beifen, Zhang, Jiyan
Formato: Artigo
Idioma:Inglês
Publicado em: Nature Publishing Group 2013
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Original Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3776642/
https://ncbi.nlm.nih.gov/pubmed/23877036
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/mt.2013.90
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