S100A1 Deficiency Impairs Post-Ischemic Angiogenesis via Compromised Proangiogenic Endothelial Cell Function and Nitric Oxide Synthase Regulation

RATIONALE: Mice lacking the EF-hand Ca(2+) sensor S100A1 display endothelial dysfunction due to distorted Ca(2+) activated NO generation. OBJECTIVE: To determine the pathophysiological role of S100A1 in endothelial cell (EC) function in experimental ischemic revascularization. METHODS AND RESULTS: P...

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Main Authors: Most, Patrick, Lerchenmüller, Carolin, Rengo, Guiseppe, Mahlmann, Adrian, Ritterhoff, Julia, Rohde, David, Goodman, Chelain, Busch, Cornelius J., Laube, Felix, Heissenberg, Julian, Pleger, Sven T., Weiss, Norbert, Katus, Hugo A., Koch, Walter J., Peppel, Karsten
Formato: Artigo
Idioma:Inglês
Publicado em: 2012
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3760372/
https://ncbi.nlm.nih.gov/pubmed/23048072
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/CIRCRESAHA.112.275156
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