Delayed Inhibition of c-Jun N-Terminal Kinase Worsens Outcomes after Focal Cerebral Ischemia

The stress-activated protein kinase c-Jun N-terminal kinase (JNK) is a central regulator in neuronal death cascades. In animal models of cerebral ischemia, acute inhibition of JNK reduces infarction and improves outcomes. Recently however, emerging data suggest that many neuronal death mediators may...

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Autors principals: Murata, Yoshihiro, Fujiwara, Norio, Seo, Ji Hae, Yan, Feng, Liu, Xiangrong, Terasaki, Yasukazu, Luo, Yumin, Arai, Ken, Ji, Xunming, Lo, Eng H.
Format: Artigo
Idioma:Inglês
Publicat: Society for Neuroscience 2012
Matèries:
Brief Communications
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC3752060/
https://ncbi.nlm.nih.gov/pubmed/22699892
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.0219-12.2012
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