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Proliferation of embryonic cardiomyocytes in zebrafish requires the sodium channel scn5Lab
In mice, homozygous deletion of the cardiac sodium channel Scn5a results in defects in cardiac morphology and embryonic death before robust sodium current can be detected. In zebrafish, morpholino knockdown of cardiac sodium channel orthologs scn5Laa and scn5Lab perturbs specification of pre-cardiac...
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| Main Authors: | , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2013
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3750094/ https://ncbi.nlm.nih.gov/pubmed/23650201 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/dvg.22400 |
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