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MYC, Metabolism, Cell Growth, and Tumorigenesis

The MYC proto-oncogene is frequently activated in human cancers through a variety of mechanisms. Its deregulated expression, unconstrained by inactivation of key checkpoints, such as p53, contributes to tumorigenesis. Unlike its normal counterpart, which is restrained by negative regulators, the unl...

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Библиографические подробности
Главный автор: Dang, Chi V.
Формат: Artigo
Язык:Inglês
Опубликовано: Cold Spring Harbor Laboratory Press 2013
Предметы:
Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC3721271/
https://ncbi.nlm.nih.gov/pubmed/23906881
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1101/cshperspect.a014217
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