Corticosteroids block binding of chemotactic peptide to its receptor on granulocytes and cause disaggregation of granulocyte aggregates in vitro.

Inhibition of complement-mediated granulocyte aggregation has recently been proposed as a mechanism of action of high-dose corticosteroids in shock states. Postulating that such inhibition might be effected through alteration of receptors function, we examined the effect of methylprednisolone (MP),...

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Argitaratua izan da:	J Clin Invest
Egile Nagusiak:	Skubitz, K M, Craddock, P R, Hammerschmidt, D E, August, J T
Formatua:	Artigo
Hizkuntza:	Inglês
Argitaratua:	American Society for Clinical Investigation 1981
Gaiak:	Research Article
Sarrera elektronikoa:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC370767/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/7251857/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI110228
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