Basis for the control of purine biosynthesis by purine ribonucleotides.
An animal model was used to determine the basis for the increase in purine biosynthesis that results from hepatic depletion of purine nucleotides, such as seen in patients with type I glycogen storage disease or following fructose administration. Mice were injected intravenously with glucose or fruc...
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| Udgivet i: | J Clin Invest |
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| Principais autores: | , , , |
| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
American Society for Clinical Investigation
1981
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC370657/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/6162862/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI110150 |
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