Prx I Suppresses K-ras-Driven Lung Tumorigenesis by Opposing Redox-Sensitive ERK/Cyclin D1 Pathway

Aims: Coupled responses of mutated K-ras and oxidative stress are often an important etiological factor in non–small-cell lung cancer (NSCLC). However, relatively few studies have examined the control mechanism of oxidative stress in oncogenic K-ras-driven NSCLC progression. Here, we studied whether...

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Detalhes bibliográficos
Main Authors: Park, Young-Ho, Kim, Sun-Uk, Lee, Bo-Kyoung, Kim, Hyun-Sun, Song, In-Sung, Shin, Hye-Jun, Han, Ying-Hao, Chang, Kyu-Tae, Kim, Jin-Man, Lee, Dong-Seok, Kim, Yeul-Hong, Choi, Chang-Min, Kim, Bo-Yeon, Yu, Dae-Yeul
Formato: Artigo
Idioma:Inglês
Publicado em: Mary Ann Liebert, Inc. 2013
Assuntos:
Original Research Communications
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3704122/
https://ncbi.nlm.nih.gov/pubmed/23186333
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1089/ars.2011.4421
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