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Pancreatic β-Cell Dysfunction and Risk of New-Onset Diabetes After Kidney Transplantation

OBJECTIVE: Chronic exposure to calcineurin inhibitors and corticosteroids poses renal transplant recipients (RTR) at high risk for development of new-onset diabetes after transplantation (NODAT). Pancreatic β-cell dysfunction may be crucial to the pathophysiology of NODAT and specific markers for β-...

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Detalhes bibliográficos
Main Authors: Zelle, Dorien M., Corpeleijn, Eva, Deinum, Jolijn, Stolk, Ronald P., Gans, Rijk O.B., Navis, Gerjan, Bakker, Stephan J.L.
Formato: Artigo
Idioma:Inglês
Publicado em: American Diabetes Association 2013
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3687295/
https://ncbi.nlm.nih.gov/pubmed/23378624
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.2337/dc12-1894
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