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Chronic nicotine exposure augments renal oxidative stress and injury through transcriptional activation of p66shc
BACKGROUND: Chronic nicotine (Ch-NIC) exposure exacerbates ischemia/reperfusion (I/R)-induced oxidative stress and acute kidney injury (AKI), and mitochondrial production of reactive oxygen species (ROS) in cultured renal proximal tubule cells (RPTCs). Because Ser36-phosphorylated p66shc modulates m...
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| Hlavní autoři: | , , , |
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| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
Oxford University Press
2013
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3685305/ https://ncbi.nlm.nih.gov/pubmed/23328708 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/ndt/gfs596 |
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