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Fetal programming alters reactive oxygen species production in sheep cardiac mitochondria

Exposure to an adverse intrauterine environment is recognized as an important risk factor for the development of cardiovascular disease later in life. Although oxidative stress has been proposed as a mechanism for the fetal programming phenotype, the role of mitochondrial O(2)(•−) (superoxide radica...

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Detalhes bibliográficos
Main Authors: Von Bergen, Nicholas H., Koppenhafer, Stacia L., Spitz, Douglas R., Volk, Kenneth A., Patel, Sonali S., Roghair, Robert D., Lamb, Fred S., Segar, Jeffrey L., Scholz, Thomas D.
Formato: Artigo
Idioma:Inglês
Publicado em: 2009
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3677965/
https://ncbi.nlm.nih.gov/pubmed/19032144
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1042/CS20080474
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