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Fetal programming alters reactive oxygen species production in sheep cardiac mitochondria
Exposure to an adverse intrauterine environment is recognized as an important risk factor for the development of cardiovascular disease later in life. Although oxidative stress has been proposed as a mechanism for the fetal programming phenotype, the role of mitochondrial O(2)(•−) (superoxide radica...
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| Autores principales: | , , , , , , , , |
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| Formato: | Artigo |
| Lenguaje: | Inglês |
| Publicado: |
2009
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| Materias: | |
| Acceso en línea: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3677965/ https://ncbi.nlm.nih.gov/pubmed/19032144 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1042/CS20080474 |
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