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Protein truncation is required for the activation of the c-myb proto-oncogene.
The protein product of the v-myb oncogene of avian myeloblastosis virus, v-Myb, differs from its normal cellular counterpart, c-Myb, by (i) expression under the control of a strong viral long terminal repeat, (ii) truncation of both its amino and carboxyl termini, (iii) replacement of these termini...
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| Published in: | Mol Cell Biol |
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| Main Authors: | , , |
| Format: | Artigo |
| Language: | Inglês |
| Published: |
Taylor & Francis
1991
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| Subjects: | |
| Online Access: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC361198/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2072904/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/mcb.11.8.3987 |
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