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Endothelial alkalinisation inhibits gap junction communication and endothelium-derived hyperpolarisations in mouse mesenteric arteries

Gap junctions mediate intercellular signalling in arteries and contribute to endothelium-dependent vasorelaxation, conducted vascular responses and vasomotion. Considering its putative role in vascular dysfunction, mechanistic insights regarding the control of gap junction conductivity are required....

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Bibliografiske detaljer
Main Authors: Boedtkjer, Ebbe, Kim, Sukhan, Aalkjaer, Christian
Format: Artigo
Sprog:Inglês
Udgivet: Blackwell Science Inc 2013
Fag:
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC3607165/
https://ncbi.nlm.nih.gov/pubmed/23297309
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2012.247478
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