CD36, but not G2A, modulates efferocytosis, inflammation, and fibrosis following bleomycin-induced lung injury

Macrophage G2A and CD36 lipid receptors are thought to mediate efferocytosis following tissue injury and thereby prevent excessive inflammation that could compromise tissue repair. To test this, we subjected mice lacking G2A or CD36 receptor to bleomycin-induced lung injury and measured efferocytosi...

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Bibliographische Detailangaben
Hauptverfasser: Parks, Brian W., Black, Leland L., Zimmerman, Kurt A., Metz, Allison E., Steele, Chad, Murphy-Ullrich, Joanne E., Kabarowski, Janusz H.
Format: Artigo
Sprache:Inglês
Veröffentlicht: The American Society for Biochemistry and Molecular Biology 2013
Schlagworte:
Research Articles
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC3605987/
https://ncbi.nlm.nih.gov/pubmed/23393303
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1194/jlr.M035352
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