The Influence of Flightless I on Toll-Like-Receptor-Mediated Inflammation in a Murine Model of Diabetic Wound Healing

Impaired wound healing and ulceration represent a serious complication of both type 1 and type 2 diabetes. Cytoskeletal protein Flightless I (Flii) is an important inhibitor of wound repair, and reduced Flii gene expression in fibroblasts increased migration, proliferation, and adhesion. As such it...

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Autori principali: Ruzehaji, Nadira, Mills, Stuart J., Melville, Elizabeth, Arkell, Ruth, Fitridge, Robert, Cowin, Allison J.
Natura: Artigo
Lingua:Inglês
Pubblicazione: Hindawi Publishing Corporation 2013
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Research Article
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC3595111/
https://ncbi.nlm.nih.gov/pubmed/23555084
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1155/2013/389792
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