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Phosphorylation of connexin43 on S279/282 may contribute to laminopathy-associated conduction defects
An understanding of the molecular mechanism behind the arrhythmic phenotype associated with laminopathies has yet to emerge. A-type lamins have been shown to interact and sequester activated phospho-ERK1/2(pERK1/2) at the nucleus. The gap junction protein connexin43 (Cx43) can be phosphorylated by p...
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| 主要な著者: | , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2012
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3594536/ https://ncbi.nlm.nih.gov/pubmed/23261543 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yexcr.2012.12.014 |
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