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Splicing in Caenorhabditis elegans does not require an AG at the 3' splice acceptor site.

The dinucleotide AG, found at the 3' end of virtually all eukaryotic pre-mRNA introns, is thought to be essential for splicing. Reduction-of-function mutations in two Caenorhabditis elegans genes, the receptor tyrosine kinase gene let-23 and the collagen gene dpy-10, both alter the AG at the end of...

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Bibliografiset tiedot
Julkaisussa:Mol Cell Biol
Päätekijät: Aroian, R V, Levy, A D, Koga, M, Ohshima, Y, Kramer, J M, Sternberg, P W
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Taylor & Francis 1993
Aiheet:
Linkit:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC358941/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8417357/
https://ncbi.nlm.nih.govhttps://doi.org/10.1128/mcb.13.1.626-637.1993
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