Splicing in Caenorhabditis elegans does not require an AG at the 3' splice acceptor site.
The dinucleotide AG, found at the 3' end of virtually all eukaryotic pre-mRNA introns, is thought to be essential for splicing. Reduction-of-function mutations in two Caenorhabditis elegans genes, the receptor tyrosine kinase gene let-23 and the collagen gene dpy-10, both alter the AG at the end of...
Tallennettuna:
| Julkaisussa: | Mol Cell Biol |
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| Päätekijät: | , , , , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
Taylor & Francis
1993
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC358941/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8417357/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/mcb.13.1.626-637.1993 |
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