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TGF-β induces miR-182 to sustain NF-κB activation in glioma subsets

The strength and duration of NF-κB signaling are tightly controlled by multiple negative feedback mechanisms. However, in cancer cells, these feedback loops are overridden through unclear mechanisms to sustain oncogenic activation of NF-κB signaling. Previously, we demonstrated that overexpression o...

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Detaylı Bibliyografya
Asıl Yazarlar: Song, Libing, Liu, Liping, Wu, Zhiqiang, Li, Yun, Ying, Zhe, Lin, Chuyong, Wu, Jueheng, Hu, Bo, Cheng, Shi-Yuan, Li, Mengfeng, Li, Jun
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: American Society for Clinical Investigation 2012
Konular:
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC3589141/
https://ncbi.nlm.nih.gov/pubmed/23006329
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI62339
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