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IL-22 Is Mainly Produced by IFNγ-Secreting Cells but Is Dispensable for Host Protection against Mycobacterium tuberculosis Infection

Anti-inflammatory treatment of autoimmune diseases is associated with an increased risk of reactivation tuberculosis (TB). Besides interleukin (IL-17)A, IL-22 represents a classical T helper (TH)17 cytokine and shares similar pathological effects in inflammatory diseases such as psoriasis or arthrit...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Päätekijät: Behrends, Jochen, Renauld, Jean-Christophe, Ehlers, Stefan, Hölscher, Christoph
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Public Library of Science 2013
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC3583848/
https://ncbi.nlm.nih.gov/pubmed/23460846
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0057379
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