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Propofol protects against high glucose-induced endothelial adhesion molecules expression in human umbilical vein endothelial cells
BACKGROUND: Hyperglycemia could induce oxidative stress, activate transcription factor nuclear factor kappa B (NF-κB), up-regulate expression of endothelial adhesion molecules, and lead to endothelial injury. Studies have indicated that propofol could attenuate oxidative stress and suppress NF-κB ac...
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| Main Authors: | , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
BioMed Central
2013
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3579710/ https://ncbi.nlm.nih.gov/pubmed/23311470 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1475-2840-12-13 |
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