Reperfusion-induced myocardial dysfunction is prevented by endogenous annexin-A1 and its N-terminal-derived peptide Ac-ANX-A1(2-26)

BACKGROUND AND PURPOSE: Annexin-A1 (ANX-A1) is an endogenous, glucocorticoid-regulated anti-inflammatory protein. The N-terminal-derived peptide Ac-ANX-A1(2–26) preserves cardiomyocyte viability, but the impact of ANX-A1-peptides on cardiac contractility is unknown. We now test the hypothesis that A...

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Detalhes bibliográficos
Main Authors: Qin, Chengxue, Buxton, Keith D, Pepe, Salvatore, Cao, Anh H, Venardos, Kylie, Love, Jane E, Kaye, David M, Yang, Yuan H, Morand, Eric F, Ritchie, Rebecca H
Formato: Artigo
Idioma:Inglês
Publicado em: Blackwell Publishing Ltd 2013
Assuntos:
Research Papers
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3570018/
https://ncbi.nlm.nih.gov/pubmed/22924634
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1476-5381.2012.02176.x
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