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Localized Sphingolipid Signaling at Presynaptic Terminals Is Regulated by Calcium Influx and Promotes Recruitment of Priming Factors
Activity-dependent changes in presynaptic function represent a critical mechanism by which synaptic strength is controlled. However, how changes in synaptic activity couple to presynaptic components to control synaptic vesicle release and recycling are poorly understood. Sphingosine kinase (SphK) is...
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| Autori principali: | , |
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| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
Society for Neuroscience
2012
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3545516/ https://ncbi.nlm.nih.gov/pubmed/23223309 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.2808-12.2012 |
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