Diabetes-Induced Hepatic Pathogenic Damage, Inflammation, Oxidative Stress, and Insulin Resistance Was Exacerbated in Zinc Deficient Mouse Model

Míreanna Comhchosúla

• Multiple Low-Dose Radiation Prevents Type 2 Diabetes-Induced Renal Damage through Attenuation of Dyslipidemia and Insulin Resistance and Subsequent Renal Inflammation and Oxidative Stress
  le: Shao, Minglong, et al.
  Foilsithe: (2014)
• Insulin Exacerbates Inflammation in Fibroblast-Like Synoviocytes
  le: Qiao, Li, et al.
  Foilsithe: (2020)
• The Prevention of Diabetic Cardiomyopathy by Non-Mitogenic Acidic Fibroblast Growth Factor Is Probably Mediated by the Suppression of Oxidative Stress and Damage
  le: Zhang, Chi, et al.
  Foilsithe: (2013)
• Zinc is essential for the transcription function of Nrf2 in human renal tubule cells in vitro and mouse kidney in vivo under the diabetic condition
  le: Li, Bing, et al.
  Foilsithe: (2014)
• Attenuation of hyperlipidemia- and diabetes-induced early-stage apoptosis and late-stage renal dysfunction via administration of fibroblast growth factor-21 is associated with suppression of renal inflammation.
  le: Chi Zhang, et al.
  Foilsithe: (2013-01-01)