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Protein Ubiquitination in Postsynaptic Densities After Transient Cerebral Ischemia
The mechanisms underlying neurologic deficits and delayed neuronal death after ischemia are not fully understood. In the present study, we report that transient cerebral ischemia induces accumulation of ubiquitinated proteins (ubi-proteins) in postsynaptic densities (PSDs). By immuno-electron micros...
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| Autors principals: | , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2004
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3518068/ https://ncbi.nlm.nih.gov/pubmed/15545915 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1097/01.WCB.0000136706.77918.21 |
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