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Cul4B regulates neural progenitor cell growth
BACKGROUND: Cullin ubiquitin ligases are activated via the covalent modification of Cullins by the small ubiquitin-like protein nedd8 in a process called neddylation. Genetic mutations of cullin-4b (cul4b) cause a prevalent type of X-linked intellectual disability (XLID) in males, but the physiologi...
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Asıl Yazarlar: | , , |
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Materyal Türü: | Artigo |
Dil: | Inglês |
Baskı/Yayın Bilgisi: |
BioMed Central
2012
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Konular: | |
Online Erişim: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3506489/ https://ncbi.nlm.nih.gov/pubmed/22992378 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1471-2202-13-112 |
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