Neonatal Exposure to the Cyanobacterial Toxin BMAA Induces Changes in Protein Expression and Neurodegeneration in Adult Hippocampus

The cyanobacterial toxin β-N-methylamino-l-alanine (BMAA) has been proposed to contribute to neurodegenerative disease. We have previously reported a selective uptake of BMAA in the mouse neonatal hippocampus and that exposure during the neonatal period causes learning and memory impairments in adul...

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Hauptverfasser: Karlsson, Oskar, Berg, Anna-Lena, Lindström, Anna-Karin, Hanrieder, Jörg, Arnerup, Gunnel, Roman, Erika, Bergquist, Jonas, Lindquist, Nils Gunnar, Brittebo, Eva B., Andersson, Malin
Format: Artigo
Sprache:Inglês
Veröffentlicht: Oxford University Press 2012
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Research Article
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC3498744/
https://ncbi.nlm.nih.gov/pubmed/22872059
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/toxsci/kfs241
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