Complement activation is not required for obliterative airway disease induced by antibodies to MHC Class I: Implications for Chronic Lung Rejection

Benzer Materyaller

• Antibodies to MHC Class II Molecules Induce Autoimmunity: Critical Role for Macrophages in the Immunopathogenesis of Obliterative Airway Disease
  Yazar:: Takenaka, Masashi, ve diğerleri
  Baskı/Yayın Bilgisi: (2012)
• Inhibition of renin angiotensin aldosterone system causes abrogation of obliterative airways disease through inhibition of tumor necrosis factor-α–dependant interleukin-17
  Yazar:: Weber, Joseph, ve diğerleri
  Baskı/Yayın Bilgisi: (2012)
• Anti-MHC induced obliterative airway disease: Selective role for CD4 and CD8 T cells in inducing immune responses to self-antigens
  Yazar:: Tiriveedhi, Venkataswarup, ve diğerleri
  Baskı/Yayın Bilgisi: (2013)
• Critical Role for IL-17A/F in the Immunopathogenesis of Obliterative Airway Disease Induced by Anti-MHC I Antibodies
  Yazar:: Basha, Haseeb Ilias, ve diğerleri
  Baskı/Yayın Bilgisi: (2013)
• Role of defensins in the pathogenesis of chronic lung allograft rejection
  Yazar:: Tiriveedhi, Venkataswarup, ve diğerleri
  Baskı/Yayın Bilgisi: (2013)