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Anti-tumor necrosis factor therapy improves insulin resistance, beta cell function and insulin signaling in active rheumatoid arthritis patients with high insulin resistance

INTRODUCTION: Prevalence of insulin resistance and the metabolic syndrome has been reported to be high in rheumatoid arthritis (RA) patients. Tumor necrosis factor (TNF), a pro-inflammatory cytokine with a major pathogenetic role in RA, may promote insulin resistance by inducing Ser(312 )phosphoryla...

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Κύριοι συγγραφείς:	Stagakis, Ilias, Bertsias, George, Karvounaris, Stylianos, Kavousanaki, Melina, Virla, Dimitra, Raptopoulou, Amalia, Kardassis, Dimitrios, Boumpas, Dimitrios T, Sidiropoulos, Prodromos I
Μορφή:	Artigo
Γλώσσα:	Inglês
Έκδοση:	BioMed Central 2012
Θέματα:	Research Article
Διαθέσιμο Online:	https://ncbi.nlm.nih.gov/pmc/articles/PMC3446524/ https://ncbi.nlm.nih.gov/pubmed/22691241 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/ar3874
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Anti-tumor necrosis factor therapy improves insulin resistance, beta cell function and insulin signaling in active rheumatoid arthritis patients with high insulin resistance

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