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Touch responsiveness in zebrafish requires voltage-gated calcium channel 2.1b
The molecular and physiological basis of the touch-unresponsive zebrafish mutant fakir has remained elusive. Here we report that the fakir phenotype is caused by a missense mutation in the gene encoding voltage-gated calcium channel 2.1b (CACNA1Ab). Injection of RNA encoding wild-type CaV2.1 restore...
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| Huvudupphovsmän: | , , , , , |
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| Materialtyp: | Artigo |
| Språk: | Inglês |
| Publicerad: |
American Physiological Society
2012
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| Ämnen: | |
| Länkar: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3434603/ https://ncbi.nlm.nih.gov/pubmed/22490555 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/jn.00839.2011 |
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