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Repression of miR-142 by p300 and MAPK is required for survival signalling via gp130 during adaptive hypertrophy
An increase in cardiac workload, ultimately resulting in hypertrophy, generates oxidative stress and therefore requires the activation of both survival and growth signal pathways. Here, we wanted to characterize the regulators, targets and mechanistic roles of miR-142, a microRNA (miRNA) negatively...
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Auteurs principaux: | , , , , , |
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Format: | Artigo |
Langue: | Inglês |
Publié: |
WILEY-VCH Verlag
2012
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Sujets: | |
Accès en ligne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3407949/ https://ncbi.nlm.nih.gov/pubmed/22367739 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/emmm.201200234 |
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