Nuclear matrix associated DNA is preferentially repaired in normal human fibroblasts, exposed to a low dose of ultraviolet light but not in Cockayne's syndrome fibroblasts.

समान संसाधन

• Deficient repair of the transcribed strand of active genes in Cockayne's syndrome cells.
  द्वारा: van Hoffen, A, और अन्य
  प्रकाशित: (1993)
• The genetic defect in Cockayne syndrome is associated with a defect in repair of UV-induced DNA damage in transcriptionally active DNA.
  द्वारा: Venema, J, और अन्य
  प्रकाशित: (1990)
• The residual repair capacity of xeroderma pigmentosum complementation group C fibroblasts is highly specific for transcriptionally active DNA.
  द्वारा: Venema, J, और अन्य
  प्रकाशित: (1990)
• The sensitivity of Cockayne's syndrome cells to DNA-damaging agents is not due to defective transcription-coupled repair of active genes.
  द्वारा: van Oosterwijk, M F, और अन्य
  प्रकाशित: (1996)
• Translocation of Cockayne syndrome group A protein to the nuclear matrix: Possible relevance to transcription-coupled DNA repair
  द्वारा: Kamiuchi, Shinya, और अन्य
  प्रकाशित: (2002)