Cell survival, DNA damage, and oncogenic transformation after a transient and reversible apoptotic response

Apoptosis serves as a protective mechanism by eliminating damaged cells through programmed cell death. After apoptotic cells pass critical checkpoints, including mitochondrial fragmentation, executioner caspase activation, and DNA damage, it is assumed that cell death inevitably follows. However, th...

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Hlavní autoři: Tang, Ho Lam, Tang, Ho Man, Mak, Keng Hou, Hu, Shaomin, Wang, Shan Shan, Wong, Kit Man, Wong, Chung Sing Timothy, Wu, Hoi Yan, Law, Hiu Tung, Liu, Kan, Talbot, C. Conover, Lau, Wan Keung, Montell, Denise J., Fung, Ming Chiu
Médium: Artigo
Jazyk:Inglês
Vydáno: The American Society for Cell Biology 2012
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On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC3374744/
https://ncbi.nlm.nih.gov/pubmed/22535522
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1091/mbc.E11-11-0926
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