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Atrial-selective prolongation of refractory period with AVE0118 is due principally to inhibition of sodium channel activity
AVE0118’s action to prolong effective refractory period (ERP) in atria but not ventricles is thought to be due to its inhibition of I(Kur). However, in non-remodeled atria, AVE0118 prolongs ERP but not action potential duration (APD(70-90)), which can be explained with inhibition of sodium, but not...
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| Main Authors: | , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2012
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3369013/ https://ncbi.nlm.nih.gov/pubmed/22370957 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1097/FJC.0b013e31824e1b93 |
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