Insufficient autophagy relates to mitochondrial dysfunction, organ failure and adverse outcome in an animal model of critical illness

Liknande verk

• Fibroblast growth factor 21 response to critical illness: effect of blood glucose control and relation with mitochondrial dysfunction, the integrated stress response and survival
  av: Thiessen, S, et al.
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• Insufficient activation of autophagy allows accumulation of cellular damage and may contribute to sustained organ failure in prolonged critically ill patients
  av: Gunst, J, et al.
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• Hepatic mitochondrial dysfunction in fluid-resuscitated porcine septic shock
  av: Wauters, J, et al.
  Publicerad: (2007)
• Impact of early versus late parenteral nutrition on morphological and molecular markers of atrophy and autophagy in skeletal muscle of critically ill patients
  av: Vanhorebeek, I, et al.
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• Receptor for advanced glycation end products axis in critically ill patients
  av: Ingels, C, et al.
  Publicerad: (2014)