A comparison of the potential role of the tetrodotoxin-insensitive sodium channels, PN3/SNS and NaN/SNS2, in rat models of chronic pain

Alterations in sodium channel expression and function have been suggested as a key molecular event underlying the abnormal processing of pain after peripheral nerve or tissue injury. Although the relative contribution of individual sodium channel subtypes to this process is unclear, the biophysical...

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Detaylı Bibliyografya
Yayımlandı:	Proc Natl Acad Sci U S A
Asıl Yazarlar:	Porreca, Frank, Lai, Josephine, Bian, Di, Wegert, Sandra, Ossipov, Michael H., Eglen, Richard M., Kassotakis, Laura, Novakovic, Sanja, Rabert, Douglas K., Sangameswaran, Lakshmi, Hunter, John C.
Materyal Türü:	Artigo
Dil:	Inglês
Baskı/Yayın Bilgisi:	National Academy of Sciences 1999
Konular:	Colloquium Paper
Online Erişim:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC33594/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/10393873/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.96.14.7640
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A comparison of the potential role of the tetrodotoxin-insensitive sodium channels, PN3/SNS and NaN/SNS2, in rat models of chronic pain

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