טוען...
Neuronal adenosine release, and not astrocytic ATP release, mediates feedback inhibition of excitatory activity
Adenosine is a potent anticonvulsant acting on excitatory synapses through A1 receptors. Cellular release of ATP, and its subsequent extracellular enzymatic degradation to adenosine, could provide a powerful mechanism for astrocytes to control the activity of neural networks during high-intensity ac...
שמור ב:
| Main Authors: | , , , , , , , |
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| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
National Academy of Sciences
2012
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3341061/ https://ncbi.nlm.nih.gov/pubmed/22421436 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1120997109 |
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