טוען...
Modulation of Gain-of-function α6*-Nicotinic Acetylcholine Receptor by β3 Subunits
We previously have shown that β3 subunits either eliminate (e.g. for all-human (h) or all-mouse (m) α6β4β3-nAChR) or potentiate (e.g. for hybrid mα6hβ4hβ3- or mα6mβ4hβ3-nAChR containing subunits from different species) function of α6*-nAChR expressed in Xenopus oocytes, and that nAChR hα6 subunit re...
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| Main Authors: | , |
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| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
American Society for Biochemistry and Molecular Biology
2012
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3340145/ https://ncbi.nlm.nih.gov/pubmed/22315221 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M111.322610 |
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