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A C-Terminal Heat Shock Protein 90 Inhibitor Decreases Hyperglycemia-induced Oxidative Stress and Improves Mitochondrial Bioenergetics in Sensory Neurons

Diabetic peripheral neuropathy (DPN) is a common complication of diabetes in which hyperglycemia-induced mitochondrial dysfunction and enhanced oxidative stress contribute to sensory neuron pathology. KU-32 is a novobiocin-based, C-terminal inhibitor of the molecular chaperone, heat shock protein 90...

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Main Authors: Zhang, Liang, Zhao, Huiping, Blagg, Brian S.J., Dobrowsky, Rick T.
Formato: Artigo
Idioma:Inglês
Publicado: 2012
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Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC3329766/
https://ncbi.nlm.nih.gov/pubmed/22413817
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1021/pr300056m
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