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PGE(2) produced by the lung augments the effector phase of allergic inflammation

Elevated PGE(2) is a hallmark of most inflammatory lesions. This lipid mediator can induce the cardinal signs of inflammation, and the beneficial actions of non-steroidal anti-inflammatory drugs are attributed to inhibition of cyclooxygenase COX-1 and COX-2, enzymes essential in the biosynthesis of...

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Detalhes bibliográficos
Main Authors: Church, Rachel J., Jania, Leigh A., Koller, Beverly H.
Formato: Artigo
Idioma:Inglês
Publicado em: 2012
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3324636/
https://ncbi.nlm.nih.gov/pubmed/22412193
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1101873
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