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Rescue of neuronal migration deficits in a mouse model of fetal Minamata disease by increasing neuronal Ca(2+) spike frequency
In the brains of patients with fetal Minamata disease (FMD), which is caused by exposure to methylmercury (MeHg) during development, many neurons are hypoplastic, ectopic, and disoriented, indicating disrupted migration, maturation, and growth. MeHg affects a myriad of signaling molecules, but littl...
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| Main Authors: | , , , , , , , , |
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| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
National Academy of Sciences
2012
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| Teme: | |
| Online dostop: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3323999/ https://ncbi.nlm.nih.gov/pubmed/22411806 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1120747109 |
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