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Mutation of zebrafish dihydrolipoamide branched-chain transacylase E2 results in motor dysfunction and models maple syrup urine disease

Analysis of zebrafish mutants that demonstrate abnormal locomotive behavior can elucidate the molecular requirements for neural network function and provide new models of human disease. Here, we show that zebrafish quetschkommode (que) mutant larvae exhibit a progressive locomotor defect that culmin...

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Detalhes bibliográficos
Main Authors: Friedrich, Timo, Lambert, Aaron M., Masino, Mark A., Downes, Gerald B.
Formato: Artigo
Idioma:Inglês
Publicado em: The Company of Biologists Limited 2012
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3291646/
https://ncbi.nlm.nih.gov/pubmed/22046030
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1242/dmm.008383
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