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Increased EID1 Nuclear Translocation Impairs Synaptic Plasticity and Memory Function Associated with Pathogenesis of Alzheimer’s disease
Though loss of function in CBP/p300, a family of CREB-binding proteins, has been causally associated with a variety of human neurological disorders, such as Rubinstein-Taybi syndrome, Huntington’s disease and drug addiction, the role of EP300 interacting inhibitor of differentiation 1 (EID1), a CBP/...
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| Main Authors: | , , , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2011
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3276739/ https://ncbi.nlm.nih.gov/pubmed/22186421 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.nbd.2011.12.007 |
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