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Retention and extinction of delay eyeblink conditioning are modulated by central cannabinoids

Rats administered the cannabinoid agonist WIN55,212-2 or the antagonist SR141716A exhibit marked deficits during acquisition of delay eyeblink conditioning, as noted by Steinmetz and Freeman in an earlier study. However, the effects of these drugs on retention and extinction of eyeblink conditioning...

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Autori principali: Steinmetz, Adam B., Freeman, John H.
Natura: Artigo
Lingua:Inglês
Pubblicazione: Cold Spring Harbor Laboratory Press 2011
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC3256566/
https://ncbi.nlm.nih.gov/pubmed/21940395
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1101/lm.2254111
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