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Retention and extinction of delay eyeblink conditioning are modulated by central cannabinoids
Rats administered the cannabinoid agonist WIN55,212-2 or the antagonist SR141716A exhibit marked deficits during acquisition of delay eyeblink conditioning, as noted by Steinmetz and Freeman in an earlier study. However, the effects of these drugs on retention and extinction of eyeblink conditioning...
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| Main Authors: | , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado: |
Cold Spring Harbor Laboratory Press
2011
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| Acceso en liña: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3256566/ https://ncbi.nlm.nih.gov/pubmed/21940395 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1101/lm.2254111 |
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