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Retention and extinction of delay eyeblink conditioning are modulated by central cannabinoids

Rats administered the cannabinoid agonist WIN55,212-2 or the antagonist SR141716A exhibit marked deficits during acquisition of delay eyeblink conditioning, as noted by Steinmetz and Freeman in an earlier study. However, the effects of these drugs on retention and extinction of eyeblink conditioning...

Deskribapen osoa

Gorde:
Xehetasun bibliografikoak
Egile Nagusiak: Steinmetz, Adam B., Freeman, John H.
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: Cold Spring Harbor Laboratory Press 2011
Gaiak:
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC3256566/
https://ncbi.nlm.nih.gov/pubmed/21940395
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1101/lm.2254111
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