Deletion of a single allele of the Pex11β gene is sufficient to cause oxidative stress, delayed differentiation and neuronal death in mouse brain

Lignende værker

• New insights into the distribution, protein abundance and subcellular localisation of the endogenous peroxisomal biogenesis proteins PEX3 and PEX19 in different organs and cell types of the adult mouse
  af: Colasante, Claudia, et al.
  Udgivet: (2017)
• PEX11β Deficiency Is Lethal and Impairs Neuronal Migration but Does Not Abrogate Peroxisome Function
  af: Li, Xiaoling, et al.
  Udgivet: (2002)
• PEX13 deficiency in mouse brain as a model of Zellweger syndrome: abnormal cerebellum formation, reactive gliosis and oxidative stress
  af: Müller, C. Catharina, et al.
  Udgivet: (2011)
• Analysis of the Level of Plasmid-Derived mRNA in the Presence of Residual Plasmid DNA by Two-Step Quantitative RT-PCR
  af: Ahlemeyer, Barbara, et al.
  Udgivet: (2020)
• Inflammatory Responses Are Not Sufficient to Cause Delayed Neuronal Death in ATP-Induced Acute Brain Injury
  af: Jeong, Hey-Kyeong, et al.
  Udgivet: (2010)