Net Intestinal Transport of Oxalate Reflects Passive Absorption and SLC26A6-mediated Secretion

Mice lacking the oxalate transporter SLC26A6 develop hyperoxalemia, hyperoxaluria, and calcium-oxalate stones as a result of a defect in intestinal oxalate secretion, but what accounts for the absorptive oxalate flux remains unknown. We measured transepithelial absorption of [(14)C]oxalate simultane...

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Bibliografiske detaljer
Main Authors: Knauf, Felix, Ko, Narae, Jiang, Zhirong, Robertson, William G., Van Itallie, Christina M., Anderson, James M., Aronson, Peter S.
Format: Artigo
Sprog:Inglês
Udgivet: American Society of Nephrology 2011
Fag:
Basic Research
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC3250206/
https://ncbi.nlm.nih.gov/pubmed/22021714
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1681/ASN.2011040433
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