Deletion of thioredoxin-interacting protein in mice impairs mitochondrial function but protects the myocardium from ischemia-reperfusion injury

Classic therapeutics for ischemic heart disease are less effective in individuals with the metabolic syndrome. As the prevalence of the metabolic syndrome is increasing, better understanding of cardiac metabolism is needed to identify potential new targets for therapeutic intervention. Thioredoxin-i...

Descrizione completa

Salvato in:
Dettagli Bibliografici
Autori principali: Yoshioka, Jun, Chutkow, William A., Lee, Samuel, Kim, Jae Bum, Yan, Jie, Tian, Rong, Lindsey, Merry L., Feener, Edward P., Seidman, Christine E., Seidman, Jonathan G., Lee, Richard T.
Natura: Artigo
Lingua:Inglês
Pubblicazione: American Society for Clinical Investigation 2011
Soggetti:
Research Article
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC3248280/
https://ncbi.nlm.nih.gov/pubmed/22201682
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI44927
Tags: Aggiungi Tag
Nessun Tag, puoi essere il primo ad aggiungerne! !

Documenti analoghi