Deletion of thioredoxin-interacting protein in mice impairs mitochondrial function but protects the myocardium from ischemia-reperfusion injury

Classic therapeutics for ischemic heart disease are less effective in individuals with the metabolic syndrome. As the prevalence of the metabolic syndrome is increasing, better understanding of cardiac metabolism is needed to identify potential new targets for therapeutic intervention. Thioredoxin-i...

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Detalhes bibliográficos
Main Authors: Yoshioka, Jun, Chutkow, William A., Lee, Samuel, Kim, Jae Bum, Yan, Jie, Tian, Rong, Lindsey, Merry L., Feener, Edward P., Seidman, Christine E., Seidman, Jonathan G., Lee, Richard T.
Formato: Artigo
Idioma:Inglês
Publicado em: American Society for Clinical Investigation 2011
Assuntos:
Research Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3248280/
https://ncbi.nlm.nih.gov/pubmed/22201682
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI44927
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