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Nse1-dependent recruitment of Smc5/6 to lesion-containing loci contributes to the repair defects of mutant complexes

Of the three structural maintenance of chromosomes (SMC) complexes, Smc5/6 remains the most poorly understood. Genetic studies have shown that Smc5/6 mutants are defective in homologous recombination (HR), and consistent with this, Smc5/6 is enriched at lesions. However, Smc5/6 is essential for viab...

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Detalhes bibliográficos
Main Authors: Tapia-Alveal, Claudia, O'Connell, Matthew J.
Formato: Artigo
Idioma:Inglês
Publicado em: The American Society for Cell Biology 2011
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3226483/
https://ncbi.nlm.nih.gov/pubmed/21976700
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1091/mbc.E11-03-0272
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